N-Acetylcholinesterase-Induced Apoptosis in Alzheimer's Disease
2008
Background
Alzheimer's disease (AD) involves loss of cholinergic neurons and Tau protein hyper-phosphorylation. Here, we report that overexpression of an N-terminally extended “synaptic” acetylcholinesterase variant, N-AChE-S is causally involved in both these phenomena.
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