Патогенетические механизмы формирования посттравматической головной боли

Pathogenetic mechanisms of posttraumatic headache (PTH) and remote results of craniocerebral injury have been considered in the paper. Complex examination of 114 patients of the main group and 87 of the control was performed. Clinicoroentgenological, neurophysiological, neurovisual investigation was used; vegetative and psychological tests were conducted. Reliable increase in magistral vascular tonus of the head and morphological changes in the form of thickening of intima-media layer was noted in the remote period of craniocerebral injuries. Stable angiospasm of arterioles and middle arteries occurs twofold more often (76,4%) in craniocerebral consequences than among patients with the other origin headache (32,3%). In neurovisual investigation, 37% of the main group patients had liquor-cystic, cystogliosis changes and hydrocephalic signs which appeared three times rarely (12%) in the control group of patients with nontraumatic cephalgias. Among PTH patients, situational and personal anxiety was less pronounced than in those with other type headaches. Posttraumatic headache is to a greater extent conditioned by magistral vessel, medulla, cervical organic changes and vegetative disorders.