Abstract 14162: NLRP3 Inflammasome Activation Initiated Through Ca2+/Calmodulin-Dependent Protein Kinase II (CaMKIIδ) Signaling in Cardiomyocytes is Essential for Pressure Overload-Induced Adverse Cardiac Remodeling

Takeshi Suetomi,Andrew Willeford,Cameron S. Brand,Shigeki Miyamoto,Joan Heller Brown

Abstract 14162: NLRP3 Inflammasome Activation Initiated Through Ca2+/Calmodulin-Dependent Protein Kinase II (CaMKIIδ) Signaling in Cardiomyocytes is Essential for Pressure Overload-Induced Adverse Cardiac Remodeling

2018

Takeshi Suetomi
Andrew Willeford
Cameron S. Brand
Shigeki Miyamoto
Joan Heller Brown

Introduction and Hypothesis: Inflammation is associated with cardiac remodeling in response to pressure overload, but how it is initiated in the absence of cell death is not known. We tested the hy...

Keywords:

Internal medicine
Heart failure
Pressure overload
Pharmacology
ca2 calmodulin
Diabetes mellitus
Protein kinase II
Programmed cell death
Medicine
Cardiology
Inflammation
NLRP3 inflammasome activation

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