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PS

P Solal-Céligny

Université de Bordeaux

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Co-Authors

Nicole Brousse

Hôpital Necker-Enfants Malades

Michel Peuchmaur

Hôpital Robert-Debré

Pierre Soubeyran

Institut Bergonié

D Émilie

Inserm

Nöel Milpied

Centre Hospitalier Universitaire de Bordeaux

Pierre Galanaud

Inserm

Philippe Colombat

Université de Tours

Corinne Haïoun

Université Paris-Est Créteil

Philippe Gaulard

Hôpitaux Universitaires Henri-Mondor

Peter Smith

University of Southampton

Cooperative Institutions

Inserm

Université Paris Cité

Assistance Publique – Hôpitaux de Paris

Centre National de la Recherche Scientifique

Hôpital Saint-Louis

Sorbonne Paris Cité

Sorbonne Université

Hôpital Necker-Enfants Malades

Centre Hospitalier Universitaire de Rennes

Université de Rennes

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IL-2 mRNA expression in Tac-positive malignant lymphomas.

Michel Peuchmaur D Émilie Marie‐Claude Crevon P Solal-Céligny M.C. Maillot

Mixed lymphocyte reaction

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Citations (56)

[Follicular lymphomas. Therapeutic indications].

P Solal-Céligny

Follicular lymphoma

Follicular Cyst

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Citations (0)

[Transmission of plasmodium falciparum malaria by syringe in a patient with heroinism].

R Bastin G Charmot I Brumpt Philippe Sansonetti P Solal-Céligny

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Citations (2)

[Follicular lymphomas: function and expression of bcl-2 gene].

Philippe Gaulard D'Agay Mf Michel Peuchmaur Nicole Brousse C Gisselbrecht

Follicular lymphoma

Expression (computer science)

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Cancer treatment with Staphylococcus aureus protein A.

P Solal-Céligny J. Simeon Albeiro López Herrera P Boivin

Extracorporeal

Protein A

Cancer Treatment

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Citations (7)

[Interstitial lymphoid pneumonia and polyadenopathies in patients infected with the LAV/HTLV III virus].

Couderc Lj Pierre‐Louis Hervé P Solal-Céligny David L. Herman D'Agay Mf

Follicular hyperplasia

Interstitial pneumonitis

Lymphoid hyperplasia

Infiltration (HVAC)

Usual interstitial pneumonia

Pneumonitis

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Citations (10)

Follicular lymphoma international prognostic index.

P Solal-Céligny Pascal Roy Philippe Colombat Joanne White Jamés O. Armitage

Follicular lymphoma

International Prognostic Index

10.1182/blood-2003-12-4434

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Citations (1,571)

C3b inactivator deficiency with immune complex manifestations.

P Solal-Céligny Michel Laviolette Jacques Hébert P ATKINS M Sirois

We report a complete C3b inactivator deficiency in a 28-year-old patient referred for bronchiectasis and recurrent pneumonia. In addition to these recurrent infections, previously described with this deficiency, he also had several immune complex manifestations (rheumatoid factor, circulating immune complexes and one episode of serum sickness). The consequences of C3b inactivator deficiency on complement activation, chemotaxis and opsonization, clearance of immune complexes, and on red blood cell sensitization are discussed. The study of the parents showed an autosomal dominant transmission.

Antibody opsonization

Complement deficiency

Rheumatoid factor

Immune complex

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Citations (31)

Iconographies supplémentaires de l'article : Efficacité du Rituximab au cours d'une granulomatose lymphomatoïde

C. Moudir-Thomas A. Foulet-Roge M. Plat R. Kaswin P. Lepic

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[Complement, granulocytes and pulmonary capillaries].

P Solal-Céligny Meyohas Mc

The activation, aggregation and adhesion of polymorphonuclear leukocytes on the endothelial wall of the pulmonary capillaries are responsible for lesions of variable severity on the alveolar-capillary membrane. The mechanism and the contribution of this leukocyte aggregation was studied to explain the respiratory abnormalities observed during the course of haemodialysis, leukapharesis and the end of extra-corporeal circulation. The activation of complement, by the formation of C5a is probably the initiator of this phenomenon. Based on these experimental models, the varied steps in the intra-capillary aggregation of leukocytes is envisaged as an explanation for the lesions in the acute respiratory distress syndrome, alveolitis caused by immune complexes and pulmonary toxicity caused by oxygen. Corticosteroids may inhibit leukocyte aggregation under certain conditions. These studies allow for a more precise pathophysiological approach for the use of steroids in their disorders.

Pathophysiology

Vascular permeability

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Citations (2)