J Vetulani

Maj Institute of Pharmacology

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Lucyna Antkiewicz‐Michaluk

Polish Academy of Sciences

Irena Nalepa

Polish Academy of Sciences

Jerzy Michaluk

Maj Institute of Pharmacology

M Sansone

Istituto Psicoanalitico per le Ricerche Sociali

Irena Romańska

Polish Academy of Sciences

Piotr Popik

Maj Institute of Pharmacology

M Melzacka

Polish Academy of Sciences

Andrzej Pilc

Polish Academy of Sciences

Marta Kowalska

Medical University of Lublin

Krystyna Reichenberg

Polish Academy of Sciences

Cooperative Institutions

Polish Academy of Sciences

Maj Institute of Pharmacology

New York University Press

Cambridge University Press

Jagiellonian University

Institute of Pharmacology

Istituto Psicoanalitico per le Ricerche Sociali

University School of Physical Education in Kraków

AGH University of Krakow

University of Wrocław

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Comparison of effectiveness of two schedules of rapid transcranial magnetic stimulation on enhancement of responsiveness to apomorphine.

PubMed (1999)

Tomasz Zyss Jacek Mamczarz Adam Roman J Vetulani

Locomotor response to apomorphine (0.5 mg/kg s.c.) was tested in rats pre-treated with multiple rapid transcranial magnetic stimulation (rTMS) or electroconvulsive shock (ECS). rTMS (B = 1.6T, f = 20 Hz, t = 300 s) was delivered 9 times during 18 days (group rTMS1, total number of stimulations 54,000) or 18 times during 18 days (group rTMS2, total number of stimulations 108,000); ECS (I = 150 mA, f = 50 Hz, t = 500 ms) was delivered 9 times at 48 h intervals. Apomorphine given 24 h after the last treatment induced hyperactivity that was significantly more pronounced in rats receiving ECS and rTMS. The effect was most pronounced for electroconvulsive treatment, the least in the rTMS1 group. The results indicate that an increase in the total number of impulses during the rTMS may increase the responsiveness of rats to dopaminergic stimulation to the level comparable with that induced by ECS.

Apomorphine

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Does Ca2+ channel blockade modulate the antidepressant-induced changes in mechanisms of adrenergic transduction?

Journal of Neural Transmission (1997)

Irena Nalepa Marta Kowalska Grzegorz Kreiner J Vetulani

10.1007/bf01277670

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Citations (9)

[Neuroleptics--phenothiazine and butyrophenone derivatives].

PubMed (1969)

J Vetulani

Butyrophenone

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Further dissociation between the memory-facilitating effects of low and memory-attenuating effects of high doses of oxytocin. A structure-activity study

Acta Neurobiologiae Experimentalis (1992)

Piotr Popik J M Ree van J Vetulani

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Modification of morphine analgesia, tolerance and abstinence by 1,2,3,4-tetrahydroisoquinoline

European Neuropsychopharmacology (2003)

J Vetulani Lucyna Antkiewicz‐Michaluk Jerzy Michaluk

Tetrahydroisoquinoline

10.1016/s0924-977x(03)90034-4

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Citations (4)

Regulacja przekaznictwa neuronalnego

Wszechświat (1997)

J Vetulani

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The effect of calcium channel blockade on the action of chronic ECT and imipramine on responses of alpha 1- and beta-adrenoceptors in the rat cerebral cortex.

PubMed (1993)

Irena Nalepa J Vetulani

Chronic co-administration of nifedipine and ECT or imipramine results in an increase in responsiveness of cerebral cortical alpha 1-adrenoceptors as measured by accumulation of inositol phosphate in cortical slices after noradrenaline stimulation; the responsiveness of beta-adrenoceptor, measured by accumulation of cyclic AMP, was depressed similarly by antidepressant treatment with and without nifedipine.

Alpha (finance)

BETA (programming language)

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Paroxetine pretreatment does not affect the behavioral and biochemical effects of chronic electroconvulsive shock in rats

European Neuropsychopharmacology (2002)

Irena Nalepa Adam Roman Magdalena Kowalska J Vetulani

Paroxetine

Affect

10.1016/s0924-977x(02)80158-4

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The action of neuroleptics in rats with altered catecholamine metabolism. 3. The effect of chlorpromazine, haloperidol and thioridazine on the brain dopamine and serotonin contents in the rats pretreated with MAO inhibitors.

PubMed (1973)

J Vetulani

Thioridazine

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The effect of drugs on accumulation of exogenous norepinephrine in nervous tissue cultured in vitro. IV. The effect of clonidine.

PubMed (1981)

H Szydłowska Z Kowalska J Vetulani

Explants of nervous tissue taken up from the area of locus coeruleus from rats 3-36 days old were cultured in vitro for 15 days, and than accumulation of norepinephrine (3 X 10-8M) was studied with and without preincubation with equimolar concentration of clonidine, using histofluorescence methods. Preincubation with clonidine increased the fluorescence in neural cells, particularly those derived from young animals, in which the pattern of fluorescence appeared to resemble the observed without preincubation only in cells from older animals in addition to fluorescence in the cells, fluorescent granules appeared in neuronal processes and in structures resembling non adrenergic nerve endings. Clonidine completely inhibited norepinephrine uptake by glial cells from very young animals, and inhibited that in the glia in colonies derived from 18-24 days old rats. However, clonidine preincubation restored accumulation of norepinephrine by oligodendroglia from 36 days old rats already lost by this age, but did not affect astrocytes.

Locus coeruleus

Nervous tissue

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