H Haas

Ruhr University Bochum

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Document Type

Co-Authors

V V Kakkar

Chitkara University

Dietmar Schulte

Ruhr University Bochum

Christoph Koban

Ruhr University Bochum

Ulrike Willutzki

Witten/Herdecke University

Barbara Neumann

Ruhr University Bochum

Holger Heine

German Center for Lung Research

Holger Petering

University of Hildesheim

Wolfgang Henke

ASML (Netherlands)

Harald Renz

Philipps University of Marburg

Angelika B. Reske‐Kunz

University Medical Center of the Johannes Gutenberg University Mainz

Cooperative Institutions

Charité - Universitätsmedizin Berlin

Research Center Borstel - Leibniz Lung Center

University of Freiburg

Technical University of Munich

Medizinische Hochschule Hannover

Ludwig-Maximilians-Universität München

German Center for Lung Research

Humboldt-Universität zu Berlin

University Medical Center Freiburg

Helmholtz Zentrum München

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A double-blind trial of suloctidil v. Placebo in intermittent claudication

British journal of surgery (1982)

N A G Jones H Haas J. Zahavi V V Kakkar

Abstract In a recent double-blind trial lasting over 6 months, 40 patients suffering from intermittent claudication were randomly allocated to receive 300 mg of suloctidil per day or exactly matching placebo capsules. In addition to treadmill walking distance, other objective criteria including ankle blood pressure response and muscle blood flow measured by 133Xe clearance were used to assess the effectiveness of therapy. Nine patients (4 in the suloctidil group and 5 controls) did not complete the trial according to the protocol. Of the remaining 31 patients, 17 were in the control group and 14 received suloctidil. A significant improvement in the absolute walking distance, the level of β-thromboglobulin (βTG) compared to pre-therapy value and the time for the ankle pressure index to return to the pre-exercise value was observed in patients receiving suloctidil.

Intermittent claudication

Claudication

Treadmill

10.1002/bjs.1800690113

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Citations (31)

[Thrombopoiesis and blood coagulation in pediatric patients with septicemia (author's transl)].

PubMed (1981)

G Mitterstieler R. Kurz H Haas G. Desoye

Thrombopoiesis

Mean platelet volume

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Experimentelle Allergologie/Immunologie

Allergo Journal (2007)

René Teich Nicole Blümer Cecilia C. Patrascan Holger Heine Otto Holst

10.1007/bf03370607

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�ber die Beeinflussung des Juckreizes durch Antistin und Aludrin

Naunyn-Schmiedeberg s Archives of Pharmacology (1949)

H Haas

10.1007/bf00244687

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Citations (1)

[Histamine and antihistaminics determination of antihistaminics].

PubMed (1951)

H Haas

Histamine H1 Antagonists

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Hereditary Antithrombin III Deficiency in an English Family

British Journal of Haematology (1981)

M F Scully H Haas Pak‐To Chan V V Kakkar

S ummary . A family including four members with decreased antithrombin III (ATIII) measured by a biological and an immunological method is described. Immunologically measured levels of other coagulation inhibitors were normal. In the antithrombin III deficient family members there was a history of thrombotic disease. Turnover of 125 I‐antithrombin III in a deficient individual showed the lower plasma levels probably to be due to a deficiency in the rate of synthesis rather than to an increased catabolic rate.

Antithrombin III deficiency

Catabolism

10.1111/j.1365-2141.1981.tb02784.x

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Citations (24)

[Burkitt's lymphoma (author's transl)].

PubMed (1976)

F. M. Schabel Ania Koller Födisch Hj H Haas Ralf Kurz

Burkitt's lymphoma

Etiology

Malignant lymphoma

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Viscosity and Red Cell Deformity in Arterial Disease

Thrombosis and Haemostasis (1979)

G Cella H Haas M.W. Rampling V V Kakkar

Haemorrheological factors have been shown to be affected in many kings of vascular disease. The present study was undertaken to correlate these factors in normal subjects and patients suffering from peripheral arterial disease. Twenty-two patients were investigated; they had moderate or severe intermittent claudication, extent of disease being confirmed by aorto-arteriography and ankle-systolic pressure studies. Twenty-five controls with no symptoms or signs of arterial disease were selected with comparable age and sex distribution. Whole blood viscosity was measured at shear rates of 230 secs-1 and 23 secs-lat 37°c using a Wells Brookfield cone plate microvisco meter. Plasma viscosity was also measured in an identical manner. Erythrocyte flexibility was measured by centrifuge technique and fibrinogen concentration as well as haematocrit by standard techniques. The fibrinogen concentration appeared to be the only significant parameter; the mean concentration in patients with peripheral vascular disease of 463 ± 73mg/l00ml in the control group ( < 0.05). Although whole blood viscosity was high in patients, when corrected to a common haematocrit, there was no significant difference between patients and controls. The same megative correlation was found for plasma viscosity. The red cell flexibility was found to be increased in patients as compared to the control group, but this effect appeared to be simply proportional to the fibrinogen concentration.

Intermittent claudication

Erythrocyte sedimentation rate

Claudication

10.1055/s-0038-1665789

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[Anti-allergic agents].

PubMed (1963)

H Haas

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