Research Article| May 01, 1947 GEOLOGIC SECTION FROM THE SIERRA NEVADA TO DEATH VALLEY, CALIFORNIA RICHARD H HOPPER RICHARD H HOPPER KEBON SIRIH 52, BATAVIA, JAVA, NETHERLANDS INDIES Search for other works by this author on: GSW Google Scholar GSA Bulletin (1947) 58 (5): 393–432. https://doi.org/10.1130/0016-7606(1947)58[393:GSFTSN]2.0.CO;2 Article history received: 02 Jun 1939 first online: 02 Mar 2017 Cite View This Citation Add to Citation Manager Share Icon Share Facebook Twitter LinkedIn MailTo Tools Icon Tools Get Permissions Search Site Citation RICHARD H HOPPER; GEOLOGIC SECTION FROM THE SIERRA NEVADA TO DEATH VALLEY, CALIFORNIA. GSA Bulletin 1947;; 58 (5): 393–432. doi: https://doi.org/10.1130/0016-7606(1947)58[393:GSFTSN]2.0.CO;2 Download citation file: Ris (Zotero) Refmanager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentBy SocietyGSA Bulletin Search Advanced Search Abstract This paper describes the geology of a part of the region between the southern Sierra Nevada and Death Valley, one of the most rugged portions of the Great Basin. The topographic features of the mountain ranges in this region strongly suggest that each range owes its present height to faulting. In the western half of the area mapped, displacements of extensive basalt flows of probable early Pleistocene age support the topographic evidences of faulting. On the summits of the ranges are areas of low relief, believed to be remnants of a single old-age erosion surface which extended across the entire region before the beginning of the range-forming fault movements. The undisturbed erosion which produced this surface ended shortly before the deposition of the fossiliferous late Pliocene or early Pleistocene Coso formation; therefore, this surface is tentatively correlated with the Ricardo erosion surface of the Mohave Desert region, which bevels tilted early Pliocene strata and which is also dislocated by range-forming faults.The ranges are composed dominantly of pre-Tertiary rocks. The pre-Cambrian metasediments, chiefly mica schists and dolomites, have an exposed thickness of 15,000 feet. Limestones, dolomites, shales, and quartzites of Paleozoic age are more than 30,000 feet thick, and fossils collected in them indicate the probable presence of all the Paleozoic systems. During the late Jurassic Nevadian orogeny the pre-Mesozoic rocks were folded, faulted, and intruded by plutonic bodies ranging from granite to gabbro. All the post-Mesozoic rocks are believed to be Miocene or younger; they include a wide variety of volcanic and sedimentary types.Most of the faulting to which the region owes its present relief occurred in the early or middle part of the Pleistocene, probably after the first (McGee) glacial stage in the Sierra Nevada. The activity on one of the major fault zones, however, has continued into the Recent epoch. All the range-forming faults whose attitudes could be determined are high-angle normal faults. This content is PDF only. Please click on the PDF icon to access. First Page Preview Close Modal You do not have access to this content, please speak to your institutional administrator if you feel you should have access.
To investigate the relationship between polymorphisms in the angiotensin converting enzyme (ACE), angiotensinogen (AGT) and type 1 angiotensin-II (AT1R) genes and (1) quantitative variations in blood pressure and (2) the blood pressure response to ACE inhibition in a hypertensive cohort.We administered monotherapy with ACE inhibitors to 125 previously untreated essential hypertensives. Genotypes for ACE insertion and deletion, AGT M235T and AT1R A1166-->C polymorphisms were determined in DNA extracted from peripheral blood leucocytes. The influence of genotype on pretreatment blood pressure and the ACE inhibitor-induced decrease in blood pressure was tested by analysis of variance and multiple regression analysis, adjusting for age, sex, body mass index, alcohol intake and, where appropriate, pretreatment blood pressure.ACE and AT1R genotypes were independent predictors of pretreatment systolic and diastolic blood pressure, with an apparent interaction between these two gene loci. Although it did not influence pretreatment blood pressure in this population, AGT genotype was an independent predictor of the blood pressure response to ACE inhibition.The ACE and AT1R gene loci (chromosomes 17q and 3q, respectively) may carry alleles influencing blood pressure variation in this hypertensive population, with a possible epistatic interaction between the two loci. The AGT T235 allele does not appear to be a marker for blood pressure variation in this group, but variants on chromosome 1q lying in or near the AGT gene may contribute to individual differences in the blood pressure response to ACE inhibition. Among essential hypertensives, differences in the ACE inhibitor response appear, in part, to be genetically determined.
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