M. García

Hospital Clínico Universitario Lozano Blesa

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F. Cambra

Hospital Universitario 12 De Octubre

M. Abradelo

Queen Elizabeth Hospital

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Álvaro García‐Sesma

Hospital Universitario 12 De Octubre

Iago Justo

Universidad Complutense de Madrid

R. Sanabria

Universidad de Costa Rica

C. Alegre

Hospital Universitario 12 De Octubre

N. Fakih

National Health Service

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Edurne Álvaro

Hospital Universitario Infanta Leonor

Alejandro Manrique

Universidad Complutense de Madrid

E. Moreno

Vall d'Hebron Hospital Universitari

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Hospital Universitario 12 De Octubre

Research Institute Hospital 12 de Octubre

Universidad Complutense de Madrid

Hospital Oncológico Docente "Conrado Benítez García"

Hospital Universitario Puerta de Hierro Majadahonda

Instituto de Salud Carlos III

Hospital Clínico San Carlos

Hospital Universitario del Henares

Universidad Autónoma de Madrid

Hospital Universitario La Paz

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6111-13. IMPACTO DE LA INSUFICIENCIA AÓRTICA EN EL PRONÓSTICO DEL SÍNDROME AÓRTICO AGUDO

Revista Española de Cardiología (2024)

Inés Ramos González-Cristóbal Carlos Ferrera Durán Ángel Fernández Pedro Muñoz Sahagún Juan González del Castillo

10.1016/s0300-8932(25)00357-4

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Decrease in thyrocalcitonin‐containing cells and analysis of other congenital anomalies in 11 patients with DiGeorge anamaly

American Journal of Medical Genetics (1993)

José Palacios Carlos Gamallo M. García José Ignacio Rodrı́guez

Abstract Experimental studies have demonstrated that the DiGeorge anomaly is due to cranial neural crest abnormalities. In the present study, the quantitation of thyrocalcitonin immunoreactive cells (C‐cells) has been used to evaluate whether cells derived from the cranial neural crest are or are not present in normal proportions in patients with this anomaly. Thyroid sections from 11 such patients and 11 control patients were studied imnmunohistochemically at autopsy in order to determine the number and distribution of thyrocalcitonin containing cells. Patients with DiGeorge anomaly showed thymic and parathyroid aplasia/hypoplasia and cardiovascular defects, such as type B interrupted aortic arch, truncus arteriosus and tetralogy of Fallot. Other associated defects were alobar holoprosencephaly and meningocels (previously unreported defects in this anomaly), arhinencephaly, renal cystic dysplasia, ureterohydronephrosis, esophageal atresia with tracheoesophageal fistula, and cleft lip and palate. The volume density of C‐cells (1.187%) and the mean number of C‐cells per follicle (1.42) was significantly lower in patients with DiGeorge anomaly than in control patients (3.475% and 2.367, respectively). These results indicate a decrease in cells derived from the neural crest in patients with DiGeorge anomaly, and support the hypothesis of a neural crest disturbance as the pathogenetic factor responsible for this anomaly. © 1993 Wiley‐Liss, Inc.

Tracheoesophageal Fistula

DiGeorge syndrome

Truncus arteriosus

10.1002/ajmg.1320460608

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Citations (35)

Coronary artery spasm following aortic valve replacement

Interactive Cardiovascular and Thoracic Surgery (2007)

Teresa Pinho Jorge Almeida M. García P Pinho

The authors present a clinical case of coronary artery spasm in a 62-year-old woman undergoing elective aortic valve replacement surgery for aortic regurgitation. On admission at ICU, the patient developed marked hemodynamic and electrical instability. Emergent coronary angiography demonstrated a pronounced spasm of the distal portion of the right coronary artery, apparently related to compression of this vessel by a chest drain tube.

10.1510/icvts.2006.145474

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Citations (9)

Liver Transplantation from Anti-Hepatitis C Virus-Positive Donors: Our Experience

Transplantation Proceedings (2012)

Edurne Álvaro M. Abradelo A Fuertes Alejandro Manrique Francisco Colina

Hepatitis C

Liver disease

Economic shortage

Viral Hepatitis

10.1016/j.transproceed.2012.05.012

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Citations (22)

Decrease in calcitonin‐containing cells in truncus arteriosus

American Journal of Medical Genetics (1993)

Carlos Gamallo M. García José Palacios José Ignacio Rodríguez

Abstract Experimental studies in chick embryos have demonstrated that truncus arteriosus (TA), a form of conotruncal cardiac defect, is due to abnormalities in the cranial neural crest. However, no data are available to support this hypothesis in humans with isolated TA. In the present study, the assessment of calcitonin immunoreactive cells (C‐cells) has been employed to evaluate whether or not the proportion of thyroid cells derived from the cranial neural crest is normal in patients with isolated TA. Thyroid sections from 15 such patients in which no other extracardiac malformations were neither clinically nor pathologically found, and from 11 control age‐matched patients were studied immunohistochemically at autopsy in order to determine the number and distribution of calcitonin‐containing cells. The volume density of C‐cells (0.888%) and the number of C‐cells per follicle (0.991) was significantly lower in patients with TA than in control patients (3.475%, and 2.367, respectively). The decrease of neural crestderived cells in the thyroid of patients with “isolated” TA documents more extent abnormalities than clinically suspected and supports the hypothesis of neural crest disturbance as the pathogenetic factor responsible for this heart malformation. © 1993 Wiley‐Liss, Inc.

Truncus arteriosus

10.1002/ajmg.1320460210

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EP09.08: Sonographic findings in fetuses affected by microdeletion 17q21.31

Ultrasound in Obstetrics and Gynecology (2016)

C. Martínez‐Payo M. García Elena Mansilla M. Ruiz de Azúa Ballesteros Pablo Lapunzina

To describe the prenatal sonographic findings in 4 fetuses affected by microdeletion 17q21.31 (Sdr. Koolen de Vries). The introduction of microarray based technology enabled genome in prenatal diagnosis resulting in the detection of recurrent microdeletions in fetuses. We try to correlation between these ultrasound alterations and Koolen-de Vries syndrome. This syndrome is characterised by developmental delay, hypotonia, facial dysmorphisms, epilepsy, heart defects and kidney/urologic anomalies. We described the ecographic findings in four fetuses affected by Koolen de Vries syndrome. An ultrasound study was performed at 12, 20–22 and 32–24 weeks of gestation. Array-SNPs was performed on uncultured amniocytes. Array-SNPs were performed by HIScan Illumina, ES CYTOSNP850k. Only one of them had an increased nuchal translucency (4.2 mm). In all of them, the sonographic study on mild gestation was normal. 3 of them had ventriculomegaly mild to moderate in the third trimester, one of them presented a corpus callosum dysgenetic and growth restriction. The mild to moderate ventriculomegaly was the most constant ecographic finding in this syndrome. Only in the third trimester was possible to suspect this fetal anomaly.

10.1002/uog.16897

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Acute Abdomen in the Geriatric Patient

International Journal of Medical Science and Clinical Research Studies (2024)

Gustavo Alejandro Paniagua Frausto Soto Durán Esther Danae Donovan Diego Ruiz Baltazar Marı́a Serrano H Reyes

Introduction Geriatric emergency department visits are most often for acute abdominal pain. More elderly patients with acute abdominal pain visit the ER as the elderly population grows. However, emergency physicians face more challenges. Elderly abdominal pain symptoms vary. However, disease manifestations, cognitive issues, and communication issues may delay diagnosis in elderly patients, increasing mortality. Indeed, early and accurate acute abdomen diagnosis in elderly patients greatly affects their outcomes.

Acute abdomen

10.47191/ijmscrs/v4-i11-23

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SCHU-06: Neuroendocrine Carcinoma of the Prostate: A Poor Prognosis?

Urology (2008)

A. Fonerón Octavio Castillo M. García M. Feria M. González

Neuroendocrine carcinoma

Prostate carcinoma

10.1016/j.urology.2008.08.013

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Arterial embolization does not decrease morbidity after distal pancreatectomy plus celiac trunk resection: A Spanish muticentre study

HPB (2020)

José Manuel Ramia E. Vicente Fernando Pardo Lídia Sabater Santiago López‐Ben

Distal pancreatectomy

10.1016/j.hpb.2020.04.059

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Current Treatment on Compartment Syndrome of the Hand

International Journal of Medical Science and Clinical Research Studies (2024)

Katia Esthefany Hernández Delgado José Emiliano González Flores H Reyes Alfonso Sandoval Polito N. Hernández

Compartment syndrome is a critical condition characterized by elevated pressures within constricted myofascial compartments, leading to vascular compromise, hypoxia, and potential irreversible injury. While commonly affecting the forearm and lower leg, compartment syndrome of the hand is rare and poses unique diagnostic and therapeutic challenges. The pathophysiology involves increased intracompartmental pressure from various etiologies, such as trauma, edema, or external compression, culminating in a cycle of ischemia and worsening edema. Rhabdomyolysis, defined by rapid muscle tissue degradation and release of intracellular components into the bloodstream, is intricately linked to compartment syndrome, with each condition potentially exacerbating the other. Elevated serum creatine kinase and myoglobin levels are hallmark diagnostic markers of rhabdomyolysis. Hand compartment syndrome, despite its rarity, necessitates prompt identification and surgical intervention. The hand's complex anatomical structure comprises ten compartments, each at risk for ischemic injury under increased pressure. Clinically, hand compartment syndrome manifests as severe pain disproportionate to injury, edema, and abnormal posturing. Diagnosis relies on clinical suspicion, supported by intracompartmental pressure monitoring. Emergent fasciotomy remains the definitive treatment, requiring multiple incisions to decompress affected compartments. Magnetic resonance imaging (MRI) is valuable for assessing tissue damage and guiding management strategies. This review underscores the importance of early recognition and timely intervention in compartment syndrome and highlights the interdependent relationship between compartment syndrome and rhabdomyolysis.

Fasciotomy

Compartment (ship)

Compartment Syndromes

10.47191/ijmscrs/v4-i12-44

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