Silvia Babini

Hospital Privado de Comunidad

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M A León Regidor

Hospital Privado de Comunidad

Federico Ceccato

Centro Científico Tecnológico - Santa Fe

Oscar Rillo

Hospital General de Agudos Dr. TEODORO ALVAREZ

Claudia Uña

Argerich Hospital

J. A. Maldonado Cocco

Instituto Nacional de Rehabilitación Psicofísica

Sergio Paira

Hospital Provincial de Rosario

Osvaldo García‐Morteo

University of Buenos Aires

Osvaldo García Morteo

Elabora Consultoria (Brazil)

Maria Alícia Lázaro

Kamuzu Central Hospital

Javier Sebastiani

Hospital Privado de Comunidad

Cooperative Institutions

Hospital Privado de Comunidad

Instituto Nacional de Rehabilitación Psicofísica

University of Buenos Aires

Hospital de Clínicas "José de San Martín"

Hospital Privado

Fundación para la Lucha contra las Enfermedades Neurológicas de la Infancia

Hospital Italiano de Buenos Aires

Argerich Hospital

Icahn School of Medicine at Mount Sinai

Universidad de Guadalajara

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[Oncogenic osteomalacia. Report of two cases].

PubMed (2015)

Fernando Jerkovich Daniel Moncet Silvia Babini Jorge Zoppi Fabiana G. Graciolli

Oncogenic osteomalacia is a rare disease. It is caused by a tumor that produces fibroblast growth factor 23, a hormone that decreases the tubular phosphate reabsorption and impairs renal hydroxylation of vitamin D. This leads to hyperphosphaturia with hypophosphatemia and low calcitriol levels. About 337 cases have been reported and we studied two cases; 44 and 70 year-old men who sought medical attention complaining of suffering diffuse bone pain over a period of approximately one year. In both cases, a laboratory test showed biochemical alterations compatible with a hypophosphatemic osteomalacia. In the first case, a soft tissue tumor of the right foot was removed, one year after the diagnosis. The patient was allowed to diminish the phosphate intake, but symptoms reappeared at this time. Eight years later, a local recurrence of the tumor was noted. A complete excision was now performed. The patient was able to finally interrupt the phosphate intake. In the second case, an F-18 fluorodeoxyglucose positron emission tomography, with computed tomography revealed a 2.26 cm diameter hypermetabolic nodule in the soft tissue of the right forefoot. After its removal, the patient discontinued the phosphate intake. Both patients are asymptomatic and show a regular phosphocalcic laboratory evaluation. The histopathological diagnosis was, in both cases, a phosphaturic mesenchymal tumor, a mixed connective tissue variant. This is the prototypical variant of these tumors.

Osteomalacia

Hypophosphatemia

Bone pain

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Citations (8)

Autoimmune interstitial lung disease in Latin-America

Clinical Immunology (2018)

Florencia Vivero Federico Campins Diana Lancellotti Pablo Malfante Silvia Babini

Usual interstitial pneumonia

10.1016/j.clim.2018.12.013

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Enfermedades que simulan polimialgia reumática

Reumatología Clínica (2011)

Federico Ceccato Claudia Uña M A León Regidor Oscar Rillo Silvia Babini

10.1016/j.reuma.2010.09.001

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Citations (17)

Clinical features of ankylosing spondylitis in women and men and its relationship with age of onset.

PubMed (1985)

Maldonado-Cocco Ja Silvia Babini Osvaldo García‐Morteo

Spondylitis

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Clinical and Serologic Characteristics of Patients with Overlap Syndrome

Medicine (1989)

María Alicia Lázaro J. A. Maldonado Cocco Luís J. Catoggio Silvia Babini Osvaldo Daniel Messina

LÁZARO, MARÍA A. M.D.; MALDONADO COCCO, JOSÉ A. M.D.; CATOGGIO, LUIS J. M.D.; BABINI, SILVIA M. M.D.; MESSINA, OSVALDO D. M.D.; MORTEO, OSVALDO GARCÍA M.D. Author Information

Extractable nuclear antigens

10.1097/00005792-198901000-00004

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Citations (60)

Conditions mimicking polymyalgia rheumatica

Reumatología Clínica (English Edition) (2011)

Federico Ceccato Claudia Uña M A León Regidor Oscar Rillo Silvia Babini

Polymyalgia rheumatica

myalgia

Erythrocyte sedimentation rate

10.1016/s2173-5743(11)70034-x

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Citations (25)

Session 4: Methodology for the assessment of treatments in hand osteoarthritis

Osteoarthritis and Cartilage (2000)

Xavier Chevalier O. Mejjad Silvia Babini

10.1053/joca.2000.0341

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Citations (13)

Atlantoaxial subluxation in systemic lupus erythematosus: further evidence of tendinous alterations.

PubMed (1990)

Silvia Babini J. A. Maldonado Cocco J C Babini M. de la Sota A Arturi

We prospectively determined the frequency of atlantoaxial subluxation in a group of patients with systemic lupus erythematosus (SLE) and analyzed its relationship with tendinous laxity, Jaccoud's syndrome and other features of the disease. Five of 59 patients (8.5%) had atlantoaxial subluxation. No patient presented atlantoaxial subluxation in neutral lateral cervical radiographs but all 5 had anterior atlantoaxial subluxation in full flexion films; one patient also had lateral subluxation. The 5 patients with atlantoaxial subluxation were compared with the remaining 54. Mean SLE disease duration was longer in patients with atlantoaxial subluxation (12 years) than in those without (6.6 years) (p less than 0.01). Jaccoud's syndrome, patellar tendon elongation and articular hypermobility were significantly more frequent in patients with atlantoaxial subluxation. The presence or history of arthritis failed to distinguish patients with and without atlantoaxial subluxation, while chronic renal failure and increased serum parathyroid hormone levels were significantly associated to the presence of atlantoaxial subluxation. We suggest that atlantoaxial subluxation is further evidence of tendinous alterations seen in patients with SLE.

Subluxation

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Citations (32)