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Ricardo Prague (Czechia)

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Deusch

Rostocker Zentrum zur Erforschung des Demografischen Wandels

Charles S. Hodges

North Carolina State University

Souza

Society of Thoracic Surgeons

Rui Rui

Southwest Forestry University

Claudia Cláudia

Bandung Institute of Technology

Freudenberg

Philipps University of Marburg

Seligmann

Carlos Sigueyuki Sediyama

Universidade Federal de Viçosa

Tocio Sediyama

Universidade Federal de Viçosa

Maria Fragaki

The General Hospital of Heraklion "Venizeleio-Pananio"

Cooperative Institutions

Universidade de São Paulo

Hospital de Santa Marta

Universidade Federal de São Carlos

Universidade do Porto

Hospital de São João

Universidade Federal de Viçosa

Universidade Federal de Minas Gerais

Royal Brompton Hospital

Universidade Estadual Paulista (Unesp)

Fundação Oswaldo Cruz

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Relevance of MUC1 mucin variable number of tandem repeats polymorphism in H pylori adhesion to gastric epithelial cells

世界胃肠病学杂志：英文版（电子版） (2008)

Natalia Natalia Costa Nuno Mendes Marcos Marcos

瞄准：评估双人脚踏车重复(VNTR ) 的 MUC1 粘蛋白变量数字的影响对胃的房间的 H pylori 粘附上的可变性。方法：酶连接了基于的免疫吸着剂试金(ELISA ) 粘附试金被执行测量不同 H pylori 紧张(HP26695 和 HPTx30a ) 的粘附到胃的癌房间线(GP202 和 MKN45 ) ， GP202 克隆与不同 VNTR 长度表示 recombinant MUC1。结果：粘附结果的评估显示出病原的紧张 HP26695 有的那 H pylori 一显著地更高(P < 0.05 ) 对所有房间线和克隆的粘附测试了，什么时候与非病原的紧张 HPTx30a 相比。细菌出现了一显著地更高(P < 0.05 ) 对 GP202 房间的粘附排队，当与 MKN45 相比房间排队时。而且，两紧张出现了一显著地更高(P < 0.05 ) 对有更大的 MUC1 VNTR 领域的 GP202 克隆的粘附。结论：这个工作证明 MUC1 粘蛋白可变性调节对胃的房间有约束力的 H pylori。细菌的粘附的程度取决于 MUC1 VNTR 领域的尺寸。粘附进一步依赖于细菌的致病力和胃的房间线。MUC1 粘蛋白可变性可以贡献决定胃粘膜的 H pylori 殖民。

MUC1

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Uma nova enfermidade de aciculas de Pinus Sp. no Brasil, causada por Cylindrocladium pteridis.

Charles S. Hodges Reis F.A. Ferreira

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A educação financeira como projeto pedagógico de inclusão no sistema regular de ensino

A. Gil de Paz Ferreira dos Santos Maria Marino de SantAnna Reis

Armando Gil Ferreira dos Santos Unigranrio Brasil gilarmfi@icloud.com Haydea Maria Marino de Sant Anna Reis ECELAH, Programa de Pos Graduacao em Ensino das Ciencias na Educacao Basica, Unigranrio Brasil hmaria@unigranrio.com.br Gisele Faur de Castro Catarino Programa de Pos Graduacao em Ensino das Ciencias na Educacao Basica, Unigranrio / UERJ Brasil gisellefaur@gmail.com Eline das Flores Victer Programa de Pos Graduacao em Ensino das Ciencias na Educacao Basica, Unigranrio Brasil elineflores@hotmail.com

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Men And Therapy: Comparisons Of College Men Who Have Sought Therapy And Have Not Sought Therapy

Reis Lionel Robert

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Impact of Particle Size, Thermal Processing, Fat Inclusion and Moisture Addition on Pellet Quality and Protein Solubility of Broiler Feeds

农业科学与技术：A (2013)

Keysuke Muramatsu Alex Alex Maiorka Isabel

Pellet

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Weighting nonlinearities on future high aggregate data rate PONs

European Conference on Optical Communication (2011)

Reis Neves Teixeira

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THE CURRICULUM THEORIES IN THE IMPLEMENTATION OF TOYS IN THE FIRST CHILDHOOD: CONTRIBUTIONS OF NEUROPSYCHOLOGY

Morganti Francesca Francesca Monteiro Kobayashi María do Carmo Reis

10.14572/nuances.v27i3

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Plaque remodeling in mouse atherosclerosis identified by high resolution in vivo magnetic resonance imaging (MRM).

Arteriosclerosis Thrombosis and Vascular Biology (2001)

R P Choudhury Juan Gilberto S. Aguinaldo James X. Rong Reis Julia Fallon

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Diabetic neuropathic pain： Physiopathology and treatment

世界糖尿病杂志：英文版（电子版） (2015)

Anne Schreiber Schreiber Carina Carina Nones RENATA RENATA

Diabetic neuropathy is a common complication of bothtype 1 and type 2 diabetes, which affects over 90% ofthe diabetic patients. Although pain is one of the mainsymptoms of diabetic neuropathy, its pathophysiologicalmechanisms are not yet fully known. It is widelyaccepted that the toxic effects of hyperglycemia play animportant role in the development of this complication,but several other hypotheses have been postulated.The management of diabetic neuropathic pain consistsbasically in excluding other causes of painful peripheral neuropathy, improving glycemic control as a prophylactic therapy and using medications to alleviate pain. First line drugs for pain relief include anticonvulsants, such as pregabalin and gabapentin and antidepressants, especially those that act to inhibit the reuptake of serotonin and noradrenaline. In addition, there is experimental and clinical evidence that opioids can be helpful in pain control, mainly if associated with first line drugs. Other agents, including for topical application, such as capsaicin cream and lidocaine patches, have also been proposed to be useful as adjuvants in the control of diabetic neuropathic pain, but the clinical evidence is insufficient to support their use. In conclusion, a better understanding of the mechanisms underlying diabetic neuropathic pain will contribute to the search of new therapies, but also to the improvement of the guidelines to optimize pain control with the drugs currently available.

Pregabalin

Diabetic Neuropathy

Pain ladder

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Über die Bakterienflora des Darms

Clinical and Experimental Medicine (1923)

Reis

10.1007/bf02609115

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Citations (10)