Abstract ChemInform is a weekly Abstracting Service, delivering concise information at a glance that was extracted from about 200 leading journals. To access a ChemInform Abstract of an article which was published elsewhere, please select a “Full Text” option. The original article is trackable via the “References” option.

Protecting group

10.1002/chin.200829249

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Polarized light-flavor antiquarks from Drell-Yan processes ofh+N→→l±+

Physical review. D. Particles, fields, gravitation, and cosmology/Physical review. D. Particles and fields (2003)

H. Kitagawa Y. Sakemi T. Yamanishi

We propose a formula to determine the first moment of difference between the polarized $\ifmmode \bar{u}\else \={u}\fi{}$ and $\overline{d}$ quarks in the nucleon, i.e., $\ensuremath{\Delta}\ifmmode \bar{u}\else \={u}\fi{}\ensuremath{-}\ensuremath{\Delta}\overline{d}$ from the Drell-Yan processes in collisions of unpolarized hadrons with longitudinally polarized nucleons by measuring outgoing lepton helicities. As coefficients in the differential cross section depend on the u- and d-quark numbers in the unpolarized hadron beam, the difference $\ensuremath{\Delta}\ifmmode \bar{u}\else \={u}\fi{}\ensuremath{-}\ensuremath{\Delta}\overline{d}$ can be independently tested by changing the hadron beam. Moreover, a formula for estimating the K factor in Drell-Yan processes is also suggested.

Bar (unit)

10.1103/physrevd.67.051501

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ChemInform Abstract: Reaction of Diols and Triols with Trialkyl Orthoesters: Facile One‐Pot Formation of Oxacyclic Compounds from Triols.

ChemInform (1993)

Hiromichi Fujioka H. Kitagawa M. KONDO Naoki Matsunaga Shinji Kitagaki

Abstract ChemInform is a weekly Abstracting Service, delivering concise information at a glance that was extracted from about 100 leading journals. To access a ChemInform Abstract of an article which was published elsewhere, please select a “Full Text” option. The original article is trackable via the “References” option.

10.1002/chin.199350098

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ChemInform Abstract: Asymmetric Synthesis Using C2‐Symmetric Diols: Use of (5R,6R)‐2,3‐ Diacetoxy‐5,6‐diphenyl‐1,4‐dioxane as a Chiral Synthetic Equivalent of 1,2‐Ethanediol 1,2‐Dicarbocation.

ChemInform (1996)

Hiromichi Fujioka H. Kitagawa Yasushi Nagatomi Yasuyuki Kita

10.1002/chin.199606169

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Proton and Lambda Fragmentation Functions in a Spectator Model

Progress of Theoretical Physics (2001)

H. Kitagawa Y. Sakemi

We study fragmentation functions (FFs) from a quark into a baryon, i.e. proton or Lambda (Λ), by using a spectator model that takes account of quark-antiquark pairs in a spectator. Values of model parameters are determined by adjusting to the quark distribution functions of the proton. The favored FF of the proton reproduces the semi-inclusive deep inelastic scattering data well, except for a slight overestimation. Lambda FFs are also studied.

Fragmentation

Lambda baryon

10.1143/ptp.105.751

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Electrically Active Nickel in Silicon Studied by DLTS in Several Kinds of Silicon Diodes

physica status solidi (a) (1990)

H. Kitagawa Shuji Tanaka

Electronic materials

10.1002/pssa.2211200141

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Reaction of Diols and Triols with Trialkyl Orthoesters: Facile One-Pot Formation of Oxacyclic Compounds from Triols

Heterocycles (1993)

Yasuyuki Kita Hiromichi Fujioka H. Kitagawa Michinori Kondo Naoki Matsunaga

10.3987/com-92-s(t)114

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Human pancreatic cancer cells disable function of Fas receptors at several levels in Fas signal transduction pathway

International Journal of Oncology (2001)

Ayman Elnemr Takahisa Ohta Akihiro Yachie Masato Kayahara H. Kitagawa

The aims of this study were to evaluate the functional expression of Fas receptors (Fas) in human pancreatic cancer cell lines; Capan-1, AsPC-1, BxPC-3, PANC-1, and MIA PaCa-2 and to search for the mechanisms of receptor-mediated inhibition of Fas signaling in these cells. Despite the expression of Fas receptors at considerable levels, exposure of these cells to agonistic Fas antibodies (500 ng/ml) induced only minimal apoptosis in 4 cell lines. The mechanisms allowing resistance to Fas-mediated apoptosis are complex. Using RT-PCR, we identified molecules which might counteract the apoptogenic signal at several levels of Fas signal transduction pathway. The most striking findings were the overexpression of Fas decoy receptors (DcR3), Fas associated phosphatase-1 (FAP-1), and FLICE-inhibitory protein (c-FLIP) in the resistant cell lines as well as in pancreatic cancer surgical specimens. In conclusion, pancreatic cancer cells express three molecules that can abrogate Fas function at different levels of Fas signaling cascade, resulting in resistance to Fas-mediated apoptosis, and this may promote the progression of this malignancy.

Fas ligand

Caspase 8

10.3892/ijo.18.2.311

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Citations (79)