Letters| June 16 2004 An Outbreak of Eruptive Pseudoangiomatosis-Like Lesions due to Mosquito Bites: Erythema punctatum Higuchi Subject Area: Dermatology , Immunology and Allergy Masanori Ban; Masanori Ban aDepartments of Dermatology,Hashima City Hospital, Hashima, and Search for other works by this author on: This Site PubMed Google Scholar Yoshiro Ichiki; Yoshiro Ichiki bGifu University School of Medicine, Gifu, Japan Search for other works by this author on: This Site PubMed Google Scholar Yasuo Kitajima Yasuo Kitajima bGifu University School of Medicine, Gifu, Japan Search for other works by this author on: This Site PubMed Google Scholar Dermatology (2004) 208 (4): 356–359. https://doi.org/10.1159/000077850 Article history Received: August 05 2003 Accepted: December 07 2003 Published Online: June 16 2004 Content Tools Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Facebook Twitter LinkedIn Email Tools Icon Tools Get Permissions Cite Icon Cite Search Site Citation Masanori Ban, Yoshiro Ichiki, Yasuo Kitajima; An Outbreak of Eruptive Pseudoangiomatosis-Like Lesions due to Mosquito Bites: Erythema punctatum Higuchi. Dermatology 1 June 2004; 208 (4): 356–359. https://doi.org/10.1159/000077850 Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentAll JournalsDermatology Search Advanced Search This content is only available via PDF. 2004Copyright / Drug Dosage / DisclaimerCopyright: All rights reserved. No part of this publication may be translated into other languages, reproduced or utilized in any form or by any means, electronic or mechanical, including photocopying, recording, microcopying, or by any information storage and retrieval system, without permission in writing from the publisher.Drug Dosage: The authors and the publisher have exerted every effort to ensure that drug selection and dosage set forth in this text are in accord with current recommendations and practice at the time of publication. However, in view of ongoing research, changes in government regulations, and the constant flow of information relating to drug therapy and drug reactions, the reader is urged to check the package insert for each drug for any changes in indications and dosage and for added warnings and precautions. This is particularly important when the recommended agent is a new and/or infrequently employed drug.Disclaimer: The statements, opinions and data contained in this publication are solely those of the individual authors and contributors and not of the publishers and the editor(s). The appearance of advertisements or/and product references in the publication is not a warranty, endorsement, or approval of the products or services advertised or of their effectiveness, quality or safety. The publisher and the editor(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. Article PDF first page preview Close Modal You do not currently have access to this content.
<i>Background:</i> Rudimentary polydactyly histologically reveals a marked neural proliferation in the dermis and a large number of Meissner corpuscles in the dermal papillae. <i>Objective:</i> Our study was performed to determine whether this neural proliferation is related to Merkel cells. <i>Method:</i> Using an antibody to cytokeratin 20, we examined the number and distribution of Merkel cells in 5 cases of rudimentary polydactyly, including an immature case without Meissner corpuscles. <i>Results:</i> Only the case without Meissner corpuscles had a markedly large number of Merkel cells and some dermal Merkel cells. <i>Conclusion:</i> The normal number of Merkel cells in the mature cases suggests that abundant Merkel cells may appear at first and disappear after the development of neural proliferation. Merkel cells may be associated with the generation of cutaneous nerve plexus and nerve endings in the upper dermis, and possibly with the development of Meissner corpuscles, at the early stage of rudimentary polydactyly.
Nodular-cystic fat necrosis, which is also called encapsulated fat necrosis, is a mobile subcutaneous nodule and histologically reveals fat necrosis encapsulated by fibrous connective tissue. In Japan, many cases have been reported, and there are some opinions that diabetes mellitus and systemic corticosteroid therapy cause this condition. The author found that out of 147 Japanese patients reported from 1982 up to 2015, 9 had the former and 23 received the latter. Systemic corticosteroid therapy may be responsible for nodular-cystic fat necrosis.
Abstract: Lipofibromatous hamartoma of the nerve is a very uncommon congenital tumor. An association between this condition and vascular malformations is not well known. We present an 11 ‐year‐old girl with a lipofibromatous hamartoma of the right median nerve with macrodactyly. She had small red macules on her right neck, chest, and arm, which were diagnosed clinically as port‐wine stains. The specimens of the enlarged nerves showed fibrous and fatty growth surrounding the nerve bundles and proliferation of the small veins. We suggest that this disorder can be accompanied by a vascular malformation.
A 61‐year‐old male presented with a 6‐month history of a subcutaneous nodule on the left forehead, which had gradually enlarged. He had suffered from headaches from 20 days before the first visit. His medical history included hepatitis C virus infection. He had had no history of trauma in this area. Clinical examination showed a subcutaneous soft nodule, 15 × 15 mm in size, which was adjacent to the left superficial temporal artery ( Fig. 1 ). Though this artery revealed strong pulsation, the nodule had neither pulsation nor tenderness. His headache was localized in the left temporal area. He had no complaints of fever or weakness. Clinically, these features suggested a pseudoaneurysm of the superficial temporal artery. A subcutaneous soft nodule adjacent to the superficial temporal artery image We operated on him under local anesthesia. An incision through the skin and subcutaneous fat exposed a nodule located within the temporal muscle. The fine branches of the superficial temporal artery reached out to the nodule through the muscle. After ligation of the branches, we resected the nodule surrounded by muscular tissue. Histopathology showed the proliferation of mature adipose cells embedded in muscular fibers. These cells had no nuclear pleomorphism or mitoses ( Fig. 2 ). The tumor tissue contained partial fibrosis and some muscular fiber bundles scattered between the adipose cells ( Fig. 3 ). Intramuscular lipoma within the temporal muscle was diagnosed. Proliferation of the mature adipose tissue within the muscle (hematoxylin‐eosin stain; magnification ×5.5) image Some muscle bundles scattered between adipose cells (hematoxylin‐eosin stain; magnification ×22) image His headache was reduced after the operation, and he has had no recurrence of the tumor for 16 months. The relationship between the tumor and his headache was unknown.
