D Stumpp

University of Giessen

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Susanne Rohrbach

University of Giessen

Bernd Niemann

University of Giessen

Heiko Bugger

Medical University of Graz

Benedikt Hermann Siegler

University Hospital Heidelberg

Mustafa Micoogullari

Heinrich Heine University Düsseldorf

K.‐D. Schlüter

Giessen School of Theology

Muhammad Agil

IPB University

Muhammad Aslam

University of Giessen

K.-D. Schlueter

University of Giessen

Ling Lü

Chinese Academy of Medical Sciences & Peking Union Medical College

Cooperative Institutions

University of Giessen

Giessen School of Theology

Heinrich Heine University Düsseldorf

Martin Luther University Halle-Wittenberg

Luther University

University of Würzburg

Johannes Gutenberg University Mainz

Universitätsklinikum Gießen und Marburg

Université d'Angers

Université Nantes Angers Le Mans

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Differential effects of C1q/tumor necrosis factor-related proteins on cardiomyocyte glucose metabolism

Diabetologie und Stoffwechsel (2015)

L Li D Stumpp Benedikt Hermann Siegler Bernd Niemann Susanne Rohrbach

Background: The adipose tissue-derived cytokine adiponectin plays a major role in glucose metabolism. A family of adiponectin paralogs, designated as C1q/tumor necrosis factor-alpha-related proteins (CTRPs) and comprising 15 members, has recently been discovered. While independent CTRP receptors have not been identified so far, the adiponectin receptor AdipoR1 has been suggested to be involved in CTRP signaling. However, there are no data on CTRP effects on glucose metabolism in cardiomyocytes available so far.

Carbohydrate Metabolism

Adiponectin receptor 1

10.1055/s-0035-1549598

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The adiponectin paralog CTRP9 but not CTRP7 mediates anti-oxidative effects in adult rat cardiomyocytes through an AMPK, adiponectin receptor and calreticulin dependent mechanism

Diabetologie und Stoffwechsel (2015)

L Li D Stumpp Benedikt Hermann Siegler Mustafa Micoogullari Bernd Niemann

Background: The adipokine adiponectin has direct beneficial effects on cardiomyocytes. However, obesity and ageing are characterized by an adiponectin deficit. Recently, a family of adiponectin paralogs, comprising 15 members so far, was discovered and designated as C1q/tumor necrosis factor-alpha-related proteins (CTRPs). While the protective adiponectin actions are well recognized, questions remain unanswered concerning the role of the CTRPs.

Adiponectin receptor 1

10.1055/s-0035-1549595

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The Adiponectin Paralog CTRP9 but not CTRP7 Mediates Anti-oxidative and Anti-hypertrophic Effects in Adult Rat Cardiomyocytes through an AMPK, Adiponectin Receptor and Calreticulin Dependent Mechanism

The Thoracic and Cardiovascular Surgeon (2016)

Bernd Niemann Ling Lü D Stumpp Benjamin Siegler Mustafa Micoogullari

Background: The anti-hypertrophic and anti-apoptotic actions of adiponectin are well recognized. However, questions remain unanswered concerning the role of the adiponectin paralogs, the C1q/tumor necrosis factor-α-related proteins (CTRPs), in these processes. Here, we investigated the mechanisms and signal transduction involved in the protective effects induced by CTRPs in cardiomyocytes.

Adiponectin receptor 1

10.1055/s-0036-1571621

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