Objective. To describe the periodontal condition of the elderly in Finland and its association with sociodemographic factors. Material and methods. The study was based on a subpopulation of 1460 persons (748 dentate persons) aged ≥65 years who participated in a nationally representative Health 2000 Survey in Finland. The data for this study were collected by means of an interview and a clinical oral examination. Results. Seventy-three percent of the participants in this study had gingival bleeding in at least one sextant. The proportion of participants with no teeth with deepened periodontal pockets ≥4 mm deep was 31%. Twenty-eight percent of the participants had one to three teeth with deepened periodontal pockets ≥4 mm deep, 15% had four to six and 26% had ≥7. At the individual level, a high number of teeth with deepened periodontal pockets was associated with sociodemographic factors such as high age, intermediate or higher education, and living in a rural area. Conclusion. At a national level, the greatest need for periodontal treatment presently derives from persons aged 65–74 years and/or those with low education, due to their high representation in the elderly population.
AbstractObjective: The objective of this study is to study the association of alcoholic beverages and serum gamma-glutamyltransferase (GGT) level with periodontal condition.Material and methods: Th...
Abstract Background This study investigates whether alcohol use predicts the periodontal pocket development over an 11‐year follow‐up period. Methods The study participants’ periodontal condition was examined both in the Health 2000 and 2011 Surveys (BRIF8901). Participants were aged 30 to 65 years, dentate, periodontally healthy, and did not have diabetes or rheumatoid arthritis in 2000. Periodontal outcome measures in 2011 were the number of teeth with deepened (≥4 mm) periodontal pockets and the presence of deepened periodontal pockets. The exposure in 2000 was alcohol use (amount [g/week], frequency [any alcohol or different beverages]). Incidence rate ratios (IRRs) with 95% confidence intervals (CI) were estimated using negative binomial regression model and Poisson regression model with a robust variance estimator. Results Overall, the amount of alcohol use or use over the risk limit in 2000 was inconsistently associated with the development of periodontal pockets (IRRs varied from 0.6 to 1.0). The frequency of alcohol use (any alcohol or different beverages) had an inconsistent association with the presence of periodontal pockets (IRRs varied from 0.5 to 1.2) while there was an inverse association with the number of teeth with periodontal pockets. Among smokers, there were no clear associations between any of the exposures and either of the outcomes. The same was found among non‐smokers except an inverse association was found between frequency of alcohol use and the number of teeth with periodontal pockets. Conclusion Alcohol use was not consistently associated with the periodontal pocket development over a period of 11 years.
To date, most epidemiological studies have shown a weak or moderate association between dental diseases such as periodontal infections, dental caries and tooth loss, and atherosclerotic vascular diseases. However, the nature of this association is not known; it may be due to the biological effect of oral infections on initiation or progress of atherosclerosis or it may be non-causal due to determinants in common, either biological or behavioural. Methodological shortcomings, inconsistent results and a lack of definite proof from intervention studies have led to the conclusion that causality between dental diseases and atherosclerotic vascular diseases has not been established. The aim of this study was to produce evidence on the nature of the association between dental diseases and atherosclerotic vascular diseases. The study uses data from the 1966 Birth Cohort of Northern Finland (N = 11,637). The data were collected in 1997–1998, when the cohort members had reached 31 years of age. The respondents were asked through a postal questionnaire about their oral health. In addition, respondents were asked about their general health and oral and general health habits. The response rate was 75.3%. Those who lived in Northern Finland or the capital city region were invited to clinical health examination (N = 8,463). Altogether 5,696 subjects supplied the data, representing 67.3% of those who were invited to the clinical examination. While the study showed an association of self-reported gingivitis, dental caries and tooth loss with the prevalent angina pectoris, it also showed that these self-reported dental diseases were not important determinants for elevated C-reactive protein levels. This suggests that the associations that were found between dental conditions and prevalent angina pectoris are mainly caused by factors other than biological mechanisms related to infection or inflammation. The lack of a biological explanation related to infections or inflammatory processes suggests that other biological mechanisms or biases, including confounding, should be considered as an alternative explanation. However, it must be noted that the possibility that oral infections also contribute to the development of atherosclerosis should not be rejected either.
Objective The aim was to study whether the anticholinergic burden of drugs is related to xerostomia and salivary secretion among community‐dwelling elderly people. Background Anticholinergic drugs have been shown to be a risk factor for dry mouth, but little is known about the effects of cumulative exposure to anticholinergic drugs measured by anticholinergic burden on salivary secretion or xerostomia. Methods The study population consisted of 152 community‐dwelling, dentate, non‐smoking, older people from the Oral Health Ge MS study. The data were collected by interviews and clinical examinations. Anticholinergic burden was determined using the Anticholinergic Drug Scale ( ADS ). A Poisson regression model with robust error variance was used to estimate relative risks ( RR ) with 95% confidence intervals ( CI 95%). Results Participants with a high‐anticholinergic burden ( ADS ≥ 3) were more likely to have xerostomia ( RR : 3.17; CI : 1.44‐6.96), low‐unstimulated salivary flow (<0.1 mL/min; RR : 2.31, CI : 1.22‐4.43) and low‐stimulated salivary flow (<1.0 mL/min; RR : 1.50, CI : 0.80‐2.81) compared to reference group ( ADS 0). In participants with a moderate anticholinergic burden ( ADS 1‐2), all the risk estimates for xerostomia, unstimulated and stimulated salivary secretion varied between 0.55 and 3.13. Additional adjustment for the total number of drugs, antihypertensives and sedative load caused only slight attenuation of the risk estimates. Conclusion A high‐anticholinergic burden was associated with low‐unstimulated salivary secretion and xerostomia.
