A low-cost and high-efficiency solid reaction method has been reported for the synthesis of manganese ferrite, MnFe2O4, with the spinel crystal structure, but impurity components affect the synthetic process. This work clarified the underlying reasons for the effects of SiO2 and Al2O3 on the synthesis of MnFe2O4. The synthetic MnFe2O4 polyhedral microparticles that showed normal and inverse spinel crystal structures with a saturated magnetization of 71.19 emu/g, a ratio of the saturation magnetization to residual magnetization (Ms/Mr) of 0.062 and a coercivity (Hc) of 6.50 Oe were obtained after 60 min at an oxidation roasting temperature of 1100 °C. The experimental results indicated that the tetrahedral Mn2+ ions and octahedral Mn3+ ions in the crystal structure of manganese ferrite, MnFe2O4, were replaced by tetrahedral Si2+ ions and octahedral Al3+ ions to form (Mn2+)x(Fe2+)y(Si2+)1-x-y[Fe3+]2O4 and (Mn2+)[Fe3+]2-x[Al3+]xO4, respectively. In addition, hercynite FexMn1-xAl2O4 with the spinel crystal structure and olivine MnxFe2-xSiO4 with an orthorhombic crystal structure were formed during the synthesis of manganese ferrite MnFe2O4 because some Fe2+ ions were easily replaced by Mn2+ ions to form the stable hercynite MnAl2O4 and olivine Mn2SiO4. The current results provide comprehensive insights for synthesizing manganese ferrite MnFe2O4 and continuously advancing the technical progress.
Objective
To explore the effect of the teaching method induced by nursing risks in nursing students in the Department of Radiotherapy.
Methods
Totally 30 nursing students practicing in the Department of Radiotherapy, the First Hospital of China Medical University between March 2014 and June 2015 were selected as control group, while another 30 nursing students practicing in the same department between September 2015 and December 2016 were selected as observation group by purposive sampling. The nursing students in the control group received conventional apprenticeship , while the nursing students in the observation group were taught with the teaching method induced by nursing risks. At the end of internship period, the nursing students were compared based on their performance in theory and practice tests.
Results
The scores of theory and practice tests of nursing students in the observation group were (93.7±4.2) , (92.9±3.5) , higher than that in the control group [ (83.3±3.2) , (85.2±3.3) ], the differences were statistically significant (t=2.283, 2.363; P<0.05) .
Conclusions
The teaching method induced by nursing risks, compared with traditional apprenticeship, can improve the internship effects of nursing students in the Department of Radiotherapy.
Key words:
Students, nursing; Clinical practice; Department of Radiotherapy; Nursing risks; Teaching method
The aim of this study was to analyze the role of LINC01554 in the pathogenesis of hepatocellular carcinoma (HCC) and explore the potential mechanism through which LINC01554 affects the migration and proliferation of HCC cells.LINC01554 expression in HCC tissues and its link to the prognosis of patients were analyzed by The Cancer Genome Atlas (TCGA) database. Quantitative Real Time-Polymerase Chain Reaction (qRT-PCR) was carried out to examine LINC01554 levels in 60 cases of HCC clinical tissues and HCC cell lines. Then, LINC01554 overexpression model was constructed using lentivirus in HCC cell lines. HCC proliferation and invasive ability were evaluated through Cell Counting Kit (CCK-8) and transwell tests, respectively. Furthermore, the potential action mechanism of LINC01554 was explored using bioinformatics analysis and in vitro cell experiments.Analysis of the TCGA database revealed that LINC01554 was remarkably under-expressed in HCC tissues. Decreased expression of LINC01554 predicted a poor prognosis for patients. Besides, LINC01554 overexpression markedly blunted the proliferation and migratory capacities of HCC cells. LINC01554 competed with NGFR to bind to microRNA-3681-3p, thereby providing possible mechanisms by which LINC01554 could participate in the progression of HCC.This study shows for the first time that LINC01554 modulates NGFR expression by binding to microRNA-3681-3p, thereby participating in the progression of HCC.
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