M. Vecchi

University of Milan

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Luca Elli

Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico

Gian Eugenio Tontini

Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico

Andrea Tertulliani

Istituto Nazionale di Geofisica e Vulcanologia

Roberto de Franchis

University of Milan

Antonio Rossi

Istituto Nazionale di Geofisica e Vulcanologia

Alice Scricciolo

Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico

Sergio Del Mese

Istituto Nazionale di Geofisica e Vulcanologia

Luca Pastorelli

University of Milan

Vincenza Lombardo

Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico

Emanuele Rondonotti

Ospedale Valduce

Cooperative Institutions

University of Milan

187

Istituti di Ricovero e Cura a Carattere Scientifico

147

Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico

100

Ospedale Maggiore

IRCCS Policlinico San Donato

University of Bologna

University of Padua

Università Cattolica del Sacro Cuore

Vita-Salute San Raffaele University

Istituto Nazionale di Geofisica e Vulcanologia

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Terremoti "dispersi" al confine tra Marche, Umbria e Lazio nel 1941-1943

Andrea Tertulliani Antonio Rossi Viviana Castelli M. Vecchi Flavio Gottardi

During World War II four moderately damaging earthquakes occurred in a portion of the Apennines crossed by the regional borders of Latium, Umbria and the Marches. The current parametric earthquake catalogues fail almost completely to record them. The earthquake of December 19, 1941 is well attested by historical sources but no Italian parametric earthquake catalogues ever recorded it. Those of January 16 and 29, 1943 were listed in the earliest Italian catalogues [Carrozzo et al., 1973; Postpischl, 1985] but they disappeared from the “NT generation” catalogues [Camassi and Stucchi, 1998; Working Group CPTI, 1999 and 2004], compiled with declustering procedures. The earthquake of March 25, 1943 is the only one to be recorded by current catalogues, but its parameters were assessed from a very poor macroseismic data base. Original information on these “lost” earthquakes was collected by an ongoing study of the October 3, 1943 earthquake (Southern Marches) and new epicentral parameters have been assessed for them. This experience is a reminder that earthquake catalogues can be incomplete not only in far off centuries but, sometimes, in recent years as well.

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OC.14.2 STAGING ESOPHAGEAL AND JUNCTIONAL CANCER: IS EUS AN ACCURATE TOOL IN T2 N0 PATIENTS?

Digestive and Liver Disease (2019)

Germana de Nucci Maria Chiara Petrone Gianpiero Manes Emanuele Asti M. Vecchi

10.1016/s1590-8658(19)30214-2

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Kallikrein-kinin system activation in Crohn's disease: differences in intestinal and systemic markers

The American Journal of Gastroenterology (2002)

Massimo Devani Massimo Cugno M. Vecchi Stefano Ferrero Federica Di Berardino

Kallikrein-kinin system activation in Crohn's disease: differences in intestinal and systemic markers

Intestinal Failure

10.1016/s0002-9270(02)04276-4

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Citations (30)

P. falciparum malaria recrudescence in a cancer patient.

PubMed (2009)

Emanuele Focà Roberto Zulli F. Buelli M. Vecchi Antonio Regazzoli

Persons living for long periods of time in malaria hyper-endemic areas may suffer from hyper-reactive malarial splenomegaly (HMS), a frequent cause of splenomegaly in such areas. Splenomegaly and sub-microscopic P. falciparum parasitaemia are hallmarks of HMS. Spleen has been suggested to play a protective antimalarial role and splenectomy may trigger symptomatic malaria attacks. Other causes of immune suppression may possibly reactivate latent malaria parasites. We report the case of an Italian 60-year-old male, who had spent 33 years in sub-Saharan Africa, who experienced a P. falciparum malaria attack 12 months after his return to Italy, concomitantly with a diagnosis of lung carcinoma possibly impairing his immune system.

Severe Malaria

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Citations (13)

P.1.235: SERUM OSTEOPROTEGERIN IS DECREASED BY ANTI-TNF TREATMENT AND CORRELATES TO C-REACTIVE PROTEIN LEVELS

Digestive and Liver Disease (2011)

Luca Pastorelli Emanuela Galliera Giada Dogliotti Carlo De Salvo Giulia Tosetti

10.1016/s1590-8658(11)60463-5

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Small-Bowel Involvement in Dermatitis Herperiformis and in Linear-IgA Bullous Dermatosis

Journal of Clinical Gastroenterology (1983)

R. deFranchis Massimo Primignani M. Cipolla M. Vecchi D. Agape

In 23 patients with dermatitis herpetiformic (DH) and five patients with linear-IgA bullous dermatosis (BD) we evaluated the occurrence of histologic jejunal changes and small-bowel function abnormalities. None of the patients showed clinical signs or symptoms of malabsorption. Morphological jejunal changes consistent with gluten-sensitive enteropathy were found in 82% of DH patients and in 60% of DD patients. However, DD patients showed only mild jejunal histologic abnormalities, whereas more severe jejunal lesions were found in most patients with DH. Functional tests showed a rough correlation with the severity of the jejunal lesions, being almost completely normal in BD patients and DH patients with mild intestinal damage, whereas most of DH patients with subtotal or total villous atrophy showed abnormal d-xylose tests and folic acid assays. Lactose tolerance tests (H2 breath test and blood glucose after oral lactose load) showed no correlation with the degree of jejunal damage.

Villous atrophy

Dermatitis herpetiformis

10.1097/00004836-198310000-00010

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Citations (15)

Silent coeliac disease may be the cause of chronic unexplained hypertransaminasemia

Gastroenterology (1998)

M. Vecchi E. Del Ninno Stefania Pacchetti D. Conte M. Landoni

10.1016/s0016-5085(98)85517-0

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Pathophysiology and therapeutic management of anemia in gastrointestinal disorders

Expert Review of Gastroenterology & Hepatology (2022)

Gaetano Bergamaschi Flavio Caprioli Marco Vincenzo Lenti Luca Elli Franco Radaelli

Anemia is a common complication of gastrointestinal (GI) disorders, with a prevalence up to 60% in celiac disease (CeD) and inflammatory bowel disease (IBD). Iron deficiency anemia (IDA) is the most prevalent form of anemia in these conditions, but chronic inflammation and vitamin B12 deficiency represent other common contributing mechanisms, especially in IBD.We discuss the pathogenesis of anemia in various medical GI disorders, the sometime problematic distinction between IDA, anemia of inflammation (AI) and the association of the two, and therapeutic and preventive measures that can be useful for the management of anemia in GI disorders. Unfortunately, with the exception of IDA and AI in IBD, large RCT concerning the treatment of anemia in GI disorders are lacking.Anemia management strategies in GI disorders are outlined, with a focus on the main prevention, diagnostic, and therapeutic measures. Specific problems and situations such as the role of gluten-free diet for IDA treatment in CeD, the choice between oral and parenteral supplementation of iron or vitamin B12 in carential anemias, the use of endoscopic procedures to stop bleeding in intestinal angiodysplasia and preventive/treatment strategies for NSAID-associated GI bleeding are discussed.

10.1080/17474124.2022.2089114

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Tu1648 PROMOTION OF PHYSICAL ACTIVITY WITH A MOBILE APP IS SUPERIOR TO PRESCRIPTION ALONE IN IMPROVING SYMPTOMS IN PATIENTS WITH IRRITABLE BOWEL SYNDROME: A RANDOMIZED CONTROLLED TRIAL

Gastroenterology (2024)

Andrea Costantino Tommaso Pessarelli Alberto Cremonesi Francesco Luciano Felice Cinque

Promotion (chess)

10.1016/s0016-5085(24)03585-6

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The Natural History of Ulcerative Proctitis: A Multicenter, Retrospective Study

The American Journal of Gastroenterology (2000)

Gianmichele Meucci M. Vecchi Marco Astegiano Luigi Beretta P. Cesari

OBJECTIVE: The aim of this study was to evaluate the clinical features and the long term evolution of patients with a well defined initial diagnosis of ulcerative proctitis. METHODS: Patients with an original diagnosis of ulcerative proctitis who had been seen at any of 13 institutions from 1989 to 1994 were identified. Data on disease onset and subsequent evolution were recorded. In addition, 575 patients with more extensive disease, treated in the same centers, were used as controls. RESULTS: A total of 341 patients satisfied the inclusion criteria. The percentage of smokers in these patients was slightly lower than in controls; no differences were found in the other clinical/demographic variables evaluated. A total of 273 patients entered long term follow-up (mean, 52 months). Proximal extension of the disease occurred in 74 of them (27.1%). The cumulative rate of proximal extension and of extension beyond the splenic flexure was 20% and 4% at 5 yr and 54% and 10% at 10 yr, respectively. The risk of proximal extension was higher in nonsmokers, in patients with >3 relapses/yr, and in patients needing systemic steroid or immunosuppressive treatment. Refractory disease was confirmed as an independent prognostic factor at multivariate analysis. CONCLUSIONS: Proximal extension of ulcerative proctitis is frequent and may occur even late after the original diagnosis. However, the risk of extension beyond the splenic flexure appears to be quite low. Smoking seems to be a protective factor against proximal extension, whereas refractoriness is a risk factor for proximal extension of the disease.

Splenic flexure

Refractory (planetary science)

10.1111/j.1572-0241.2000.t01-1-01770.x

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Citations (143)