Z. Adamis

National Public Health and Medical Officer Service

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Erzsébet Tátrai

Orszagos Kornyezetegeszsegugyi Intezet

G Ungváry

National Public Health and Medical Officer Service

M. Timár

University of Debrecen

Zuzana Kovačíková

Comenius University Bratislava

M Brózik

National Institute of Oncology

Judit Kárpáti

Semmelweis University

Miklós Náray

Hungarian Institute of Occupational Health

M. Szenzenstein

Koichi Honma

Kameda Medical Center

I. Apostol

Gheorghe Asachi Technical University of Iași

Cooperative Institutions

Hungarian Institute of Occupational Health

Semmelweis University

Hungarian Academy of Sciences

Dokkyo Medical University

National Institute of Occupational Health

Agricultural Biotechnology Institute

National Public Health and Medical Officer Service

HUN-REN Institute of Experimental Medicine

University of Edinburgh

Dokkyo University

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The pulmonary toxicity of cinnamon dust in rats.

PubMed (1995)

Erzsébet Tátrai Z. Adamis G Ungváry

The histopathological effect of a single intratracheal dose of respirable cinnamon dust, cinnamon dust extract, and cellulose dust on the lungs of rats was studied sequentially one, seven days and one month after treatment. Exposure to respirable cinnamon and cellulose dusts resulted in alveobronchiolitis at the end of the first and seventh day, and fibrotic changes by the end of the first month. As the extract of cinnamon dust caused no histopathological alterations, it is assumed that the cellulose content of cinnamon dust was responsible for the histological reactions.

Intratracheal instillation

Histopathological examination

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Citations (8)

Occupational exposure to organophosphorus insecticides and synthetic pyrethroid

International Archives of Occupational and Environmental Health (1985)

Z. Adamis A. Antal I. F�zesi J. Moln�r L.K. Nagy

10.1007/bf00405271

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Citations (34)

Evaluation of Dust Toxicity by Short-Term Methods

Springer eBooks (1985)

Z. Adamis Erzsébet Tátrai M. Timár G Ungváry

10.1007/978-3-642-70630-1_57

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Significance of Surface Properties of Quartz and Quartz-Containing Mineral Dusts to the Pulmonary Toxicity

Springer eBooks (1994)

Z. Adamis Erzsébet Tátrai M. Timár G Ungváry

Intratracheal instillation

10.1007/978-3-642-79041-6_38

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Surface changes in type II pneumocytes isolated from rats during the cultivation.

PubMed (1999)

Zuzana Kovačíková Erzsébet Tátrai Z. Adamis

Type II cells isolated from the rat lung were maintained in culture for 8 days. The activity of alkaline phosphatase and lectin binding properties were studied. The alkaline phosphatase activity and the number of lamellar bodies were continually decreasing during the studied time period. The profile of lectin binding (Maclura pomifera and Ricinus communis) did not change during the cultivation.

Lamellar granule

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The dermatological aspects of the use of pesticides in greenhouses.

R. Balog A. Strigens K. Gombosi N. Tòth Z. Adamis

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Role of cellulose in wood dust‐induced fibrosing alveo‐bronchiolitis in rat

Journal of Applied Toxicology (1995)

Frzsébet Tátrai Z. Adamis U Böhm K Merétey G Ungváry

Abstract Our experiments suggest that in the development of plant dust‐induced fibrosing alveobronchiolitis—Scadding's fibrosing alveolitis—the cellulose content of plant dusts has a decisive aetiological role. Namely, the wood dust (pine) and the cellulose induced morphologically identical granulomatous inflammation and fibrosis, whereas the fibre‐free extract of wood dust did not cause pathological changes in the lungs. The induction of H 2 O 2 and superoxide anion production, shown in vitro in leucocytes, probably has an important role in the development of fibrosis.

10.1002/jat.2550150110

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Citations (33)

<italic>In Vitro</italic> and <italic>in Vivo</italic> Tests for Determination of the Pathogenicity of Quartz, Diatomaceous Earth, Mordenite and Clinoptilolite

The Annals of Occupational Hygiene (2000)

Z. Adamis Erzsébet Tátrai Kenji Honma Éva Six G Ungváry

The effects of samples of crystalline quartz, diatomaceous earth, mordenite and clinoptilolite were investigated in vitro (as concerns erythrocyte haemolysis and lactate dehydrogenase (LDH) release from peritoneal macrophages) and in vivo (on LDH, protein and phospholipids in rat bronchoalveolar lavage (BAL), and phospholipids in rat lung tissue). The respirable mineral samples were instilled intratracheally. Determinations in the BAL were carried out after 15, 60 and 180 days, and in the lung tissue after 90, 180 and 360 days. Quartz DQ and quartz FQ induced acute, subacute and chronic inflammation and progressive fibrosis. However, due to the Al2O3 contamination on the surface of the particles quartz FQ caused a delayed response in vivo. Diatomaceous earth produced acute/subacute inflammation that gradually became more moderate after 60 days. Clinoptilolite was inert, whereas the other zeolite sample, mordenite, was cytotoxic in vivo. The reason for this was presumably the needle and rod-shaped particles in the mordenite samples. The investigation revealed that different in vitro and in vivo methods canprovide valuable data concerning the pulmonary toxicity of minerals.

Clinoptilolite

Mordenite

Silicosis

10.1093/annhyg/44.1.67

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Citations (39)

The effects of some dusts on suspended macrophages.

Egeszsegtudomany (1966)

Z. Adamis M. Timár

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A study on lung toxicity of respirable hard metal dusts in rats

The Annals of Occupational Hygiene (1997)

Z. Adamis Erzsébet Tátrai Kenji Honma Judit Kárpáti G Ungváry

The aetiology of hard metal lung disease has not been clarified so far. The pulmonary toxicity of respirable dusts collected in a hard metal factory was studied in vivo in rats. The effect of the samples was examined 1, 4, 7 and 30 days after single intratracheal injection. Lactate dehydrogenase (LDH), acid phosphatase (AP), protein and phospholipid were determined in cellfree bronchoalveolar lavage (BAL) and lung tissue. The lungs and regional lymph nodes were processed histologically. Lung toxicity of the samples collected during hard metal production varied. Samples containing considerable amount of cobalt dissolved upon acid treatment were found to induce inflammation. It has been established that the biological effect of samples of identical composition is changed by heat treatment and pre-sintering. Our examinations seem to prove that cobalt plays a prominent role in the development of pathological alterations.

Hard metal

Pulmonary toxicity

Silicosis

10.1016/s0003-4878(97)00014-8

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Citations (12)