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Jun Wei

Northwestern University

35

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2144

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Targeting CD38-Dependent NAD+ Metabolism to Mitigate Multiple Organ Fibrosis

2020 Social Science Research Network
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Targeting CD38-dependent NAD+ Metabolism to Mitigate Multiple Organ Fibrosis

2020 iScience
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958 Pharmacological inhibition of JAK/STAT signaling by tofacitinib prevents organ fibrosis: Novel therapy for scleroderma

2019 Journal of Investigative Dermatology
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The JAK/STAT pathway is activated in systemic sclerosis and is effectively targeted by tofacitinib:

2019
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The non-neuronal cyclin-dependent kinase 5 is a fibrotic mediator potentially implicated in systemic sclerosis and a novel therapeutic target

2017 Oncotarget
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Identification of Optimal Mouse Models of Systemic Sclerosis by Interspecies Comparative Genomics

2016 Arthritis & Rheumatism
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A synthetic PPAR-γ agonist triterpenoid ameliorates experimental fibrosis: PPAR-γ-independent suppression of fibrotic responses

2014 Annals of the Rheumatic Diseases
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Myofibroblasts in Cutaneous Fibrosis Originate from Adiponectin-Positive Intradermal Progenitors Running Head: Adipocytemyofibroblast transition (AMT) in skin fibrosis

2014
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p300 Is Elevated in Systemic Sclerosis and Its Expression Is Positively Regulated by TGF-β: Epigenetic Feed-Forward Amplification of Fibrosis

2013 Journal of Investigative Dermatology
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Early Growth Response 3 (Egr-3) Is Induced by Transforming Growth Factor-β and Regulates Fibrogenic Responses

2013 American Journal of Pathology
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Citations (35)