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David E. Moller

Merck & Co.
Harvard University
Beth Israel Deaconess Medical Center

162

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24712

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Direct AMPK Activation Corrects NASH in Rodents Through Metabolic Effects and Direct Action on Inflammation and Fibrogenesis.

2021
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Imeglimin amplifies glucose-stimulated insulin release from diabetic islets via a distinct mechanism of action.

2021 PLOS ONE
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Imeglimin preserves islet β‐cell mass in Type 2 diabetic ZDF rats

2020
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Erratum. Evaluation of Efficacy and Safety of the Glucagon Receptor Antagonist LY2409021 in Patients With Type 2 Diabetes: 12- and 24-Week Phase 2 Studies. Diabetes Care 2016;39:1241–1249

2017 Diabetes Care
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FGF21-based pharmacotherapy – potential utility for metabolic disorders

2014 Trends in Endocrinology and Metabolism
- Ruth E. Gimeno
- David E. Moller
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Rational Design of a Fibroblast Growth Factor 21-Based Clinical Candidate, LY2405319

2013 PLOS ONE
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The effects of LY2405319, an FGF21 Analog, in obese human subjects with type 2 diabetes

2013 Cell Metabolism
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Translational Medicine and Drug Discovery: OBESITY: NEW MECHANISMS AND TRANSLATIONAL PARADIGMS

2011
- Gregory Gaich
- David E. Moller
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Discovery of (2R)-2-(3-{3-[(4-Methoxyphenyl)carbonyl]-2-methyl-6-(trifluoromethoxy)-1H-indol-1-yl}phenoxy)butanoic Acid (MK-0533): A Novel Selective Peroxisome Proliferator-Activated Receptor γ Modulator for the Treatment of Type 2 Diabetes Mellitus with a Reduced Potential to Increase Plasma and Extracellular Fluid Volume

2009 Journal of Medicinal Chemistry
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O-10: Mutant insulin receptors Arg1174Gln and Pro1178Leu — naturally occurring insulin receptor mutations as tools in the dissection of insulin signal transduction pathways

2009 Experimental and Clinical Endocrinology & Diabetes
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