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Cornelius Krasel

University of Marburg

54

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Interaction between BID and VDAC1 is required for mitochondrial demise and cell death in neurons

2021 bioRxiv
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Disentangling bias between Gq, GRK2, and arrestin3 recruitment to the M3 muscarinic acetylcholine receptor.

2021 eLife
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Low intrinsic efficacy for G protein activation can explain the improved side effect profiles of new opioid agonists

2020 Science Signaling
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The Effect of Cell Surface Expression and Linker Sequence on the Recruitment of Arrestin to the GIP Receptor

2020 Frontiers in Pharmacology
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Correction: Al-Zamel, N., et al. A Dual GLP-1/GIP Receptor Agonist Does Not Antagonize Glucagon at Its Receptor but May Act as a Biased Agonist at the GLP-1 Receptor. Int. J. Mol. Sci. 2019, 20, 3532.

2020 International Journal of Molecular Sciences
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The N-termini of GRK2 and GRK3 simulate the stimulating effects of RKIP on β-adrenoceptors

2019 Biochemical and Biophysical Research Communications
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Multisite phosphorylation is required for sustained interaction with GRKs and arrestins during rapid μ-opioid receptor desensitization

2018 Science Signaling
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SK2 channels regulate mitochondrial respiration and mitochondrial Ca2+ uptake

2017 Cell Death & Differentiation
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A reverse signaling pathway downstream of Sema4A controls cell migration via Scrib

2017 Journal of Cell Biology
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Analysis of Receptor Tyrosine Kinase and G-Protein Coupled Receptor Signaling Dynamics on Micro-Structured Surfaces

2016 Biophysical Journal
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